FGF20 and Parkinson's disease: No evidence of association or pathogenicity via α‐synuclein expression
Identifieur interne : 002308 ( Main/Exploration ); précédent : 002307; suivant : 002309FGF20 and Parkinson's disease: No evidence of association or pathogenicity via α‐synuclein expression
Auteurs : Christian Wider [États-Unis] ; Justus C. Dachsel [États-Unis] ; Alexandra I. Soto [États-Unis] ; Michael G. Heckman [États-Unis] ; Nancy N. Diehl [États-Unis] ; Mei Yue [États-Unis] ; Sarah Lincoln [États-Unis] ; Jan O. Aasly [Norvège] ; Kristoffer Haugarvoll [États-Unis, Norvège] ; John Q. Trojanowski [États-Unis] ; Spiridon Papapetropoulos [États-Unis] ; Deborah Mash [États-Unis] ; Alex Rajput [Canada] ; Ali H. Rajput [Canada] ; J. Mark Gibson [Irlande (pays)] ; Timothy Lynch [Irlande (pays)] ; Dennis W. Dickson [États-Unis] ; Ryan J. Uitti [États-Unis] ; Zbigniew K. Wszolek [États-Unis] ; Matthew J. Farrer [États-Unis] ; Owen A. Ross [États-Unis]Source :
- Movement Disorders [ 0885-3185 ] ; 2009-02-15.
Descripteurs français
- Pascal (Inist)
English descriptors
- KwdEn :
- MESH :
- chemical , genetics : Fibroblast Growth Factors, alpha-Synuclein.
- genetics : Parkinson Disease.
- Adult, Aged, Aged, 80 and over, Female, Humans, Male, Middle Aged.
Abstract
Genetic variation in fibroblast growth factor 20 (FGF20) has been associated with risk of Parkinson's disease (PD). Functional evidence suggested the T allele of one SNP, rs12720208 C/T, altered PD risk by increasing FGF20 and α‐synuclein protein levels. Herein we report our association study of FGF20 and PD risk in four patient‐control series (total: 1,262 patients and 1,881 controls), and measurements of FGF20 and α‐synuclein protein levels in brain samples (nine patients). We found no evidence of association between FGF20 variability and PD risk, and no relationship between the rs12720208 genotype, FGF20 and α‐synuclein protein levels. © 2009 Movement Disorder Society
Url:
- https://api.istex.fr/document/4AD79CC8B3140A1D4F04BA5FC1D17431D1A1E3D2/fulltext/pdf
- http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2875476
DOI: 10.1002/mds.22442
Affiliations:
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Le document en format XML
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<term>Aged</term>
<term>Aged, 80 and over</term>
<term>FGF20</term>
<term>Female</term>
<term>Fibroblast Growth Factors (genetics)</term>
<term>Humans</term>
<term>Male</term>
<term>Middle Aged</term>
<term>Nervous system diseases</term>
<term>Parkinson Disease (genetics)</term>
<term>Parkinson disease</term>
<term>Parkinson's disease</term>
<term>Pathogenicity</term>
<term>alpha-Synuclein (genetics)</term>
<term>association study</term>
<term>genetics</term>
<term>α‐synuclein</term>
</keywords>
<keywords scheme="MESH" type="chemical" qualifier="genetics" xml:lang="en"><term>Fibroblast Growth Factors</term>
<term>alpha-Synuclein</term>
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</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Adult</term>
<term>Aged</term>
<term>Aged, 80 and over</term>
<term>Female</term>
<term>Humans</term>
<term>Male</term>
<term>Middle Aged</term>
</keywords>
<keywords scheme="Pascal" xml:lang="fr"><term>Maladie de Parkinson</term>
<term>Pathologie du système nerveux</term>
<term>Pouvoir pathogène</term>
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<front><div type="abstract" xml:lang="en">Genetic variation in fibroblast growth factor 20 (FGF20) has been associated with risk of Parkinson's disease (PD). Functional evidence suggested the T allele of one SNP, rs12720208 C/T, altered PD risk by increasing FGF20 and α‐synuclein protein levels. Herein we report our association study of FGF20 and PD risk in four patient‐control series (total: 1,262 patients and 1,881 controls), and measurements of FGF20 and α‐synuclein protein levels in brain samples (nine patients). We found no evidence of association between FGF20 variability and PD risk, and no relationship between the rs12720208 genotype, FGF20 and α‐synuclein protein levels. © 2009 Movement Disorder Society</div>
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<name sortKey="Lincoln, Sarah" sort="Lincoln, Sarah" uniqKey="Lincoln S" first="Sarah" last="Lincoln">Sarah Lincoln</name>
<name sortKey="Mash, Deborah" sort="Mash, Deborah" uniqKey="Mash D" first="Deborah" last="Mash">Deborah Mash</name>
<name sortKey="Papapetropoulos, Spiridon" sort="Papapetropoulos, Spiridon" uniqKey="Papapetropoulos S" first="Spiridon" last="Papapetropoulos">Spiridon Papapetropoulos</name>
<name sortKey="Ross, Owen A" sort="Ross, Owen A" uniqKey="Ross O" first="Owen A." last="Ross">Owen A. Ross</name>
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<name sortKey="Wszolek, Zbigniew K" sort="Wszolek, Zbigniew K" uniqKey="Wszolek Z" first="Zbigniew K." last="Wszolek">Zbigniew K. Wszolek</name>
<name sortKey="Yue, Mei" sort="Yue, Mei" uniqKey="Yue M" first="Mei" last="Yue">Mei Yue</name>
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<country name="Norvège"><region name="Trøndelag"><name sortKey="Aasly, Jan O" sort="Aasly, Jan O" uniqKey="Aasly J" first="Jan O." last="Aasly">Jan O. Aasly</name>
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<name sortKey="Aasly, Jan O" sort="Aasly, Jan O" uniqKey="Aasly J" first="Jan O." last="Aasly">Jan O. Aasly</name>
<name sortKey="Haugarvoll, Kristoffer" sort="Haugarvoll, Kristoffer" uniqKey="Haugarvoll K" first="Kristoffer" last="Haugarvoll">Kristoffer Haugarvoll</name>
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<country name="Canada"><noRegion><name sortKey="Rajput, Alex" sort="Rajput, Alex" uniqKey="Rajput A" first="Alex" last="Rajput">Alex Rajput</name>
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<name sortKey="Rajput, Ali H" sort="Rajput, Ali H" uniqKey="Rajput A" first="Ali H." last="Rajput">Ali H. Rajput</name>
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<country name="Irlande (pays)"><noRegion><name sortKey="Gibson, J Mark" sort="Gibson, J Mark" uniqKey="Gibson J" first="J. Mark" last="Gibson">J. Mark Gibson</name>
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<name sortKey="Lynch, Timothy" sort="Lynch, Timothy" uniqKey="Lynch T" first="Timothy" last="Lynch">Timothy Lynch</name>
</country>
</tree>
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